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Amidez (Amitriptyline)
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Amidez

Amidez works by affecting the balance of certain chemicals (such as serotonin) in the brain. It helps improve mood, relieve anxiety and tension, helps you sleep better, and increase your energy level. Amidez is readily absorbed and starts action within approximately 6 hours of taking it orally.

Other names for this medication:
Amiclozor, Amitar, Amitin, Amitone, Amitop, Amitrim, Amitrip, Amitriptyline, Amitryn, Amitwix, Amity, Amixide, Amline, Amrea, Amtipax, Amtran, Amypres, Aptric, Bluneel, Cotrip, Crypton, Emotrip, Eptanol, Estiline, Goldep, Librodep, Lodep, Mitrip, Normaline, Sarotena, Sereval, Tadamit, Tryline

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Also known as: Amitriptyline

Description

{name} (amitriptyline) is a tricyclic antidepressant with sedative effects. Amitriptyline affects certain chemical messengers (neurotransmitters) that communicate between brain cells and help regulate mood.

{name} is a prescription medicine used to treat symptoms of depression.

Dosage

Take Amidez tablets orally with water, with or without food. Take Amidez for one to four times a day at the same time. The treatment can be resulting after 4 weeks. If you want to achieve most effective results do not stop taking Amidez suddenly.

Overdose

Seek emergency medical attention or call the Poison Help line at 1-800-222-1222. An overdose of amitriptyline can be fatal.

Overdose symptoms may include irregular heart rhythm, feeling like you might pass out, seizures, or coma.

Storage

Store at room temperature between 20 and 25 degrees C (68 and 77 degrees F) away from moisture and heat. Throw away any unused medicine after the expiration date. Keep out of the reach of children.

Side effects

The most common side effects associated with Amidez are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.

Contraindications

You should not use Amidez if you are allergic to amitriptyline, or:

if you have recently had a heart attack.

Do not use Amidez if you have used an MAO inhibitor in the past 14 days. A dangerous drug interaction could occur. MAO inhibitors include isocarboxazid, linezolid, methylene blue injection, phenelzine, rasagiline, selegiline, tranylcypromine, and others.

Tell your doctor if you have used an "SSRI" antidepressant in the past 5 weeks, such as citalopram, escitalopram, fluoxetine (Prozac), fluvoxamine, paroxetine, sertraline (Zoloft), trazodone, or vilazodone.

To make sure Amidez is safe for you, tell your doctor if you have ever had:

bipolar disorder (manic-depression) or schizophrenia; mental illness or psychosis; liver disease; heart disease; a heart attack, stroke, or seizures; diabetes (amitriptyline may raise or lower blood sugar); glaucoma; or problems with urination.

Some young people have thoughts about suicide when first taking an antidepressant. Your doctor should check your progress at regular visits. Your family or other caregivers should also be alert to changes in your mood or symptoms.

Tell your doctor if you are pregnant or breastfeeding.

Amidez is not approved for use by anyone younger than 12 years old.

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Low potassium concentration in soil solution of forest soils favors radiocaesium soil-plant transfer. The maximum uptake of (18)F-deoxyglucose on positron emission tomography scan correlates with survival, hypoxia inducible factor-1alpha and GLUT-1 in non-small cell lung cancer. Silver-haired bat rabies virus (SHBRV) infection induces a strong virus-specific immune response in the periphery of the host, but death is common due to the failure to open the blood-brain barrier (BBB) and deliver immune effectors to central nervous system (CNS) tissues. Epidemiology and prevention of chronic arsenicosis: an Indian perspective. To test whether the efficacy and safety of the selective heart rate-reducing agent ivabradine changes according to age in chronic heart failure (HF) patients. Exogenous OPG blocked TRAIL-induced apoptosis, but IL-1beta induction of OPG did not influence TRAIL-induced cell death. In vitro experiments showed that NtcA binds specifically but with different affinities to two sites in the spsB promoter region. These results indicated that the RBP-J-mediated signaling might inhibit the differentiation of NSCs into INPs and support the generation of certain early born neurons at early neurogenic stages.

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