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Calcigard (Nifedipine)

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Calcigard belongs to the group of Calcium Channel Broker (CCB). This medication is used to control chest pain (angina) and hyper tension (high blood pressure). Calcigard was produced by Bayer, a reputed German pharmaceutical company. The initial studies on the drug were undertaken in the year of 1970. This drug is approved by FDA.

Other names for this medication:
Adalate, Adipine, Afeditab, Amarkor, Antrolin, Belnif, Bresben, Calcheck, Cardules, Citilat, Conducil, Corotrend, Depine, Emaberin, Fenidina, Ficor, Glopir, Herlat, Hexadilat, Jutadilat, Marivolon, Milfadin, Nicardia, Nidicard, Nifecard, Nifecardia, Nifedate, Nifedigel, Nifedin, Nifedipina, Nifedipino, Nifehexal, Nifensar, Nifeslow, Nifestad, Nifical, Nifin, Niften, Nipin, Nipress, Nycopin, Sepamit

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Also known as: Nifedipine


Nifedipine is used alone or together with other medicines to treat severe chest pain (angina) or high blood pressure (hypertension). High blood pressure adds to the workload of the heart and arteries. If it continues for a long time, the heart and arteries may not function properly. This can damage the blood vessels of the brain, heart, and kidneys, resulting in a stroke, heart failure, or kidney failure. High blood pressure may also increase the risk of heart attacks. These problems may be less likely to occur if blood pressure is controlled.

Nifedipine is a calcium channel blocker. It works by affecting the movement of calcium into the cells of the heart and blood vessels. As a result, nifedipine relaxes blood vessels and increases the supply of blood and oxygen to the heart while reducing its workload.

This medicine is available only with your doctor's prescription.


This section provides information on the proper use of a number of products that contain nifedipine. It may not be specific to Calcigard OROS. Please read with care.

In addition to the use of this medicine, treatment for your high blood pressure may include weight control and changes in the types of foods you eat, especially foods high in sodium (salt). Your doctor will tell you which of these are most important for you. You should check with your doctor before changing your diet.

Many patients who have high blood pressure will not notice any signs of the problem. In fact, many may feel normal. It is very important that you take your medicine exactly as directed and that you keep your appointments with your doctor even if you feel well.

Remember that this medicine will not cure your high blood pressure, but it does help control it. You must continue to take it as directed if you expect to lower your blood pressure and keep it down. You may have to take high blood pressure medicine for the rest of your life. If high blood pressure is not treated, it can cause serious problems such as heart failure, blood vessel disease, stroke, or kidney disease.

Swallow the extended release tablet whole. Do not break, crush, or chew it. It is best to take this tablet on an empty stomach.

If you are taking the extended-release tablets, part of the tablet may pass into your stool after your body has absorbed the medicine. This is normal and nothing to worry about.

The dose of this medicine will be different for different patients. Follow your doctor's orders or the directions on the label. The following information includes only the average doses of this medicine. If your dose is different, do not change it unless your doctor tells you to do so.

The amount of medicine that you take depends on the strength of the medicine. Also, the number of doses you take each day, the time allowed between doses, and the length of time you take the medicine depend on the medical problem for which you are using the medicine.

For treatment of chest pain or high blood pressure: For oral dosage form (capsules): Adults—At first, 10 milligrams (mg) three times a day. Your doctor may increase your dose as needed. Children—Use and dose must be determined by your doctor. For oral dosage form (extended-release tablets): Adults—At first, 30 or 60 milligrams (mg) once a day. Your doctor may increase your dose as needed. However, the dose is usually not more than 90 mg once a day. Children—Use and dose must be determined by your doctor.


If you overdose Calcigard and you don't feel good you should visit your doctor or health care provider immediately.


Store the medicine in a closed container at room temperature, away from heat, moisture, and direct light. Keep from freezing.

Keep out of the reach of children.

Do not keep outdated medicine or medicine no longer needed.

Ask your healthcare professional how you should dispose of any medicine you do not use.

Detailed Nifedipine dosage information

Side effects

The most common side effects associated with Calcigard are:

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The alphaIIbbeta3 fibrinogen receptor contains free thiols that are required for the activation of this receptor to a fibrinogen-binding conformation. Survey of iron-accumulative macroregenerative nodules in cirrhotic livers. The present study provides genetic evidence that inflammation and immune dysregulation responses that involve the interferon signaling pathway in the BM microenvironment are associated with MDS pathogenesis, which suggests BM MSCs as a possible therapeutic target in MDS. MET overexpression assessed by new interpretation method predicts gene amplification and poor survival in advanced gastric carcinomas. Batches of a million Y chromosomes were produced from each of several 40 ml donations of peripheral blood. This has led surgeons to perform hemiarthroplasty or hemiarthroplasty with biological resurfacing of the glenoid in an effort to avoid the potential problems with a polyethylene glenoid and obtain durable and acceptable results for these patients.

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In cardiomyopathy unlike coronary heart disease, right ventricular function tends to decrease and shows markedly altered links with the left heart. To determine the safety, toxicity, and maximum-tolerated dose of a sequence-specific combination of the histone deacetylase inhibitor (HDACi), valproic acid (VPA), and epirubicin in solid tumor malignancies and to define the clinical feasibility of VPA as an HDACi. This review reports recent findings on norfloxacin and tinidazole tablets uses in hindi the multiple factors that regulate skeletal muscle growth in neonates. However, the incidence as well as the severity of the instability had decreased significantly following the two operative procedures. This new method could effectively improve the detection rate of the cercarial infested water and is suitable for the low-degree infested water. General magnetic therapy in the treatment and prevention of duodenal ulcer disease. Bleomycin-induced changes in pulmonary microvascular albumin permeability and extravascular albumin space.

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Investigating brain community structure abnormalities in bipolar disorder using path length associated community estimation. hermsii a novel member of the complement regulator-acquiring surface protein (CRASP) family, designated BhCRASP-1, that binds the complement regulators factor H (FH) and FH-related protein 1 (FHR-1) but not FH-like protein 1 (FHL-1). Sustainable Management of Subterranean Termite Populations (Isoptera: Rhinotermitidae) in Armstrong Park, New Orleans, With Durable Baits. Oswestry disability index (ODI) and Short Form 36 (SF-36) values were prospectively collected preoperatively and at 1 year postoperatively. Preoperative Gelfoam tretinoin cream drug class embolization aided in the palliative debulking of the tumor at operation. Wetland drainage and agricultural transformations in the southern highlands of Papua New Guinea. Biosynthesis of an antifungal oligopeptide in Burkholderia contaminans strain MS14.

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For each number of phases, ESV was measured between aortic valve closure and mitral valve opening and normalized by reference ESV measured at 100 phases/R-R. Afferent and efferent aspects of mandibular sensorimotor control in adults who stutter. The amount of alpha 2M correlated with the degree of inflammation. This study on real-life, prospectively obtained data confirms that the PUCAI is highly feasible by virtue of the noninvasiveness, valid, and responsive index. Understanding the molecular and cellular basis of splotch NTDs, as well as the mechanisms by which the frequency of defects is influenced by genetic and environmental factors (such as sub-optimal folate status), may provide insight into the causation of these severe congenital malformations in humans. The periodicity of FC is different in DLB and VaD cases, with important implications for the underlying causal mechanisms and for differential diagnosis. Here, we have established a novel mouse model of ICC by liver-specific Kras activation and Pten deletion.

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